JAK2V617F homozygosity drives a phenotypic switch in myeloproliferative neoplasms, but is insufficient to sustain disease

Li, J.; Kent, D. G.; Godfrey, A. L.; Manning, H.; Nangalia, J.; Aziz, A.; Chen, E.; Saeb-Parsy, K.; Fink, J.; Sneade, R.; Hamilton, T. L.; Pask, D. C.; Silber, Y.; Zhao, X.; Ghevaert, C.; Liu, P.; Green, A. R.

期刊名称Blood
作者Li, J.; Kent, D. G.; Godfrey, A. L.; Manning, H.; Nangalia, J.; Aziz, A.; Chen, E.; Saeb-Parsy, K.; Fink, J.; Sneade, R.; Hamilton, T. L.; Pask, D. C.; Silber, Y.; Zhao, X.; Ghevaert, C.; Liu, P.; Green, A. R.
卷第123卷
期第20期
印刷ISSN0006-4971
电子ISSN1528-0020
文件大小2281.20KB
发表时间2014-05-15
DOI10.1182/blood-2013-06-510222
页码3139-3151

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JAK2V617F homozygosity drives a phenotypic switch in myeloproliferative neoplasms, but is insufficient to sustain disease